Endemol, Dutch production company
Tuesday, April 5, 2011
Illuminati in Advertising; Part II
Satanic Symbolism
Very blatant, the big fat satanic symbol, on the church-ALTAR, how do we know it is satanic?
Because Aleister Crowley told us so..
..And take a close look at the jewelry being worn by Bush insider Katherine Harris (pictured below). Here again is this Masonic/satanic symbolism (her pendant/necklace).
TV-evangelist Pat Robertson..
or is he..
This pose of his hand is called "the lion's paw",
this is masonic!
And again.. so the first was no coincidence..
So this is also no coincidence (horns of satan)
Christians ect cannot be freemasons, or so it has been said,
ofcourse this is BS, because secret societies most of the time
derrive from religion, a lot of religious people are masons, like
for instance Pat Robertson, Reverend Jesse Jackson and many others!
Research Link
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Sunday, April 3, 2011
First portrait of Jesus found??
The image is eerily familiar: a bearded young man with flowing curly hair. After lying for nearly 2,000 years hidden in a cave in the Holy Land, the fine detail is difficult to determine. But in a certain light it is not difficult to interpret the marks around the figure’s brow as a crown of thorns.
The extraordinary picture of one of the recently discovered hoard of up to 70 lead codices – booklets – found in a cave in the hills overlooking the Sea of Galilee is one reason Bible historians are clamouring to get their hands on the ancient artefacts.
If genuine, this could be the first-ever portrait of Jesus Christ, possibly even created in the lifetime of those who knew him
For the entire story CLICK HERE!!
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The Tillman Story;
More and more people are starting to realize that we have been lied to by our own governments, The Tillman Story is the perfect example for how cover-ups work, this is the story of Pat Tillman, an American Football talent who gave his life for a war that never should have started..
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The Freemasonic Elements in the Murdercase of JFK
You cannot escape the freemasonic elements in the case of the murder on John F. Kennedy.
For instance, the person who shot one of the most famous films of all time; Abraham Zapruder (Zapruder-film) was a 32nd degree freemason, he also had ties with C.I.A.
The person where Dealey Plaza was named after; George Bannerman Dealey, a 33rd degree freemason.
The freemasonic obelisk (which has 13 segments btw) on Dealey Plaza;
Before JFK went to Dallas (Texas) he was in Fort Worth (Texas), and there he was at a lunch and at that lunch schoolchildren sang the song "the Eyes of Texas", which is a song written by a freemason and has all the freemasonic elements (read my post on "The Eyes of Texas").
Lyndon B. Johnson, JFK's successor, was a freemason.
Earl Warren (Warren-commission) was a past Grandmaster in freemasonry, Richard B. Russell (Warren-commission) was a freemason, Gerald Ford (Warren-sommission), later president, was a freemason.
Those are "known" freemasons..
Allen W. Dulles (Warren-commission) was former director of the C.I.A, and one of the masterminds of Project Paperclip, Dulles was angry at JFK for firing him and promising him to shatter the C.I.A into a thousand pieces, Dulles was also a freemason btw.
This is the make-up of the Warren Commission, APPOINTED BY LYNDON B. JOHNSON another freemason, people who had bitter feelings for JFK, HOW CAN THIS EVER BE FAIR?!
"Freemasons are always in high positions because they know how to keep things secret" this is what DeMolay-man Bill Cooper said (DeMolday is a masonic youth society), Bill Cooper also said William (Bill) Greer, the limo-driver of the presidential limousine, had fired the fatal headshot, he also said he had inside info (documents ect) obtained at his job saying this is the factual truth, when analyzing the zapruder film you can see what Cooper said to be a gun is actually the reflection of agent Roy Kellerman's hair, sorry Bill Cooper, just saw you credibillity go down the drain..
NO GUN;
JFK wanted to "sack" 33rd degree freemason J. Edgar Hoover as the director of the F.B.I, when JFK was killed, his successor Lyndon B. Johnson (also freemason) granted fellow-mason J. Edgar Hoover director-ship of the F.B.I for life..
Dallas is located just south of the 33 degree of latitude. The 33rd degree is Freemasonry's highest rank.
Dealey Plaza is actually shaped as a pyramid, and JFk was shot at app. the "eye" of the pyramid;
Arlen Spector, the one that came up with "the magic bullet theory"(which is ofcourse nonsense) was also a 33rd degree freemason.
Kennedy's motorcade was rolling toward the "Triple Underpass" when he was slain by, according to some analysts, three gunmen. Three tramps were arrested right after the murder. Hiram Abiff, architect of Solomon's Temple and mythic progenitor of Freemasonry was murdered according to Masonic legend by three "unworthy craftsmen."
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Saturday, April 2, 2011
Most Wanted; Osama Bin Laden?
I was surfing on the net, and ended up of the fbi-page, don't ask me why, lol, it happened..
Looking through the site i clicked on the 10 most wanted list of the FBI;
I saw his pic and everything, and WHAT he is wanted for..;
NO SEPTEMBER 11TH 2001??????
Really weird.. maybe because he didn't do it, like he said he didn't?
(He said, if i remember correctly, on september 12th 2001 that he was not responsible for the wtc planecrashes ect)
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Looking through the site i clicked on the 10 most wanted list of the FBI;
I saw his pic and everything, and WHAT he is wanted for..;
(click on pic for larger image)
NO SEPTEMBER 11TH 2001??????
Really weird.. maybe because he didn't do it, like he said he didn't?
(He said, if i remember correctly, on september 12th 2001 that he was not responsible for the wtc planecrashes ect)
Leave thoughts as comments..
Friday, April 1, 2011
Poison; Monosodium Glutamate / MSG
Monosodium glutamate (MSG): a villain and promoter of liver inflammation and dysplasia.
Nakanishi Y, Tsuneyama K, Fujimoto M, Salunga TL, Nomoto K, An JL, Takano Y, Iizuka S, Nagata M, Suzuki W, Shimada T, Aburada M, Nakano M, Selmi C, Gershwin ME.
Department of Diagnostic Pathology, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama, Toyama, Japan.
Chronic inflammation is a common theme in a variety of disease pathways, including autoimmune diseases. The pathways of chronic inflammation are well illustrated by nonalcoholic steatohepatitis (NASH), which is of a serious concern due to its increasing prevalence in the westernized world and its direct correlation with lifestyle factors, particularly diet. Importantly, NASH may ultimately lead to the development of hepatocellular carcinoma. We previously reported that injection of monosodium glutamate (MSG) in ICR mice leads to the development of significant inflammation, central obesity, and type 2 diabetes. To directly address the long-term consequences of MSG on inflammation, we have performed serial analysis of MSG-injected mice and focused in particular on liver pathology. By 6 and 12 months of age, all MSG-treated mice developed NAFLD and NASH-like histology, respectively. In particular, the murine steatohepatitis at 12 months was virtually undistinguishable from human NASH. Further, dysplastic nodular lesions were detected in some cases within the fibrotic liver parenchyma. We submit that MSG treatment of mice induces obesity and diabetes with steatosis and steatohepatitis resembling human NAFLD and NASH with pre-neoplastic lesions. These results take on considerable significance in light of the widespread usage of dietary MSG and we suggest that MSG should have its safety profile re-examined and be potentially withdrawn from the food chain.
Sensory and autonomic nerve changes in the monosodium glutamate-treated rat: a model of type II diabetes.
Rats that had been injected with monosodium glutamate (MSG) neonatally were studied for up to 70 weeks and compared with age-matched control rats to study changes in glucose tolerance and in sympathetic and sensory nerves. At 61 and 65 weeks of age, there were significant differences in glucose tolerance between the MSG and control groups, and the MSG group had raised fasting blood glucose. These changes were not associated with changes in the number of beta-cells in the islets of Langerhans. In addition, the diabetic MSG-treated rats had central obesity and cataracts. Hypoalgesia to thermal stimuli was present in MSG-treated rats as early as 6 weeks and persisted at 70 weeks. However, no differences were observed in the distribution of substance P, the neurokinin-1 receptor or calcitonin gene-related peptide in the dorsal horn of L3-L5 at this age (70 weeks). Diabetic MSG-treated animals at 65 and 70 weeks of age had significantly reduced noradrenaline concentrations in the heart, tail artery and ileum, while concentrations in the adrenal gland and corpus cavernosum were significantly increased. There was also a significant increase in adrenal adrenaline, dopamine and serotonin, largely attributable to changes in weight of the adrenal gland in the MSG-treated animals. The results indicate that MSG-treated animals develop a form of type II diabetes by about 60 weeks of age, and that there are significant changes in amine levels in various tissues associated with these developments.
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Nakanishi Y, Tsuneyama K, Fujimoto M, Salunga TL, Nomoto K, An JL, Takano Y, Iizuka S, Nagata M, Suzuki W, Shimada T, Aburada M, Nakano M, Selmi C, Gershwin ME.
Department of Diagnostic Pathology, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama, Toyama, Japan.
Chronic inflammation is a common theme in a variety of disease pathways, including autoimmune diseases. The pathways of chronic inflammation are well illustrated by nonalcoholic steatohepatitis (NASH), which is of a serious concern due to its increasing prevalence in the westernized world and its direct correlation with lifestyle factors, particularly diet. Importantly, NASH may ultimately lead to the development of hepatocellular carcinoma. We previously reported that injection of monosodium glutamate (MSG) in ICR mice leads to the development of significant inflammation, central obesity, and type 2 diabetes. To directly address the long-term consequences of MSG on inflammation, we have performed serial analysis of MSG-injected mice and focused in particular on liver pathology. By 6 and 12 months of age, all MSG-treated mice developed NAFLD and NASH-like histology, respectively. In particular, the murine steatohepatitis at 12 months was virtually undistinguishable from human NASH. Further, dysplastic nodular lesions were detected in some cases within the fibrotic liver parenchyma. We submit that MSG treatment of mice induces obesity and diabetes with steatosis and steatohepatitis resembling human NAFLD and NASH with pre-neoplastic lesions. These results take on considerable significance in light of the widespread usage of dietary MSG and we suggest that MSG should have its safety profile re-examined and be potentially withdrawn from the food chain.
Sensory and autonomic nerve changes in the monosodium glutamate-treated rat: a model of type II diabetes.
Rats that had been injected with monosodium glutamate (MSG) neonatally were studied for up to 70 weeks and compared with age-matched control rats to study changes in glucose tolerance and in sympathetic and sensory nerves. At 61 and 65 weeks of age, there were significant differences in glucose tolerance between the MSG and control groups, and the MSG group had raised fasting blood glucose. These changes were not associated with changes in the number of beta-cells in the islets of Langerhans. In addition, the diabetic MSG-treated rats had central obesity and cataracts. Hypoalgesia to thermal stimuli was present in MSG-treated rats as early as 6 weeks and persisted at 70 weeks. However, no differences were observed in the distribution of substance P, the neurokinin-1 receptor or calcitonin gene-related peptide in the dorsal horn of L3-L5 at this age (70 weeks). Diabetic MSG-treated animals at 65 and 70 weeks of age had significantly reduced noradrenaline concentrations in the heart, tail artery and ileum, while concentrations in the adrenal gland and corpus cavernosum were significantly increased. There was also a significant increase in adrenal adrenaline, dopamine and serotonin, largely attributable to changes in weight of the adrenal gland in the MSG-treated animals. The results indicate that MSG-treated animals develop a form of type II diabetes by about 60 weeks of age, and that there are significant changes in amine levels in various tissues associated with these developments.
Leave thoughts as comments..
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